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Evidence profile · Growth Hormone Axis

GHRP-2

GHRP-2 is a growth hormone secretagogue. Japan approves it as a single-dose diagnostic agent.

EvidenceModerateRegulatoryApproved in Japan (diagnostic)WADAProhibited (S2.2.4)

Checked by the PU Research editorial teamevidence-graded against primary sources. Last check: 23 July 2026. Sources: 3. Who checks this

Sport prohibition notice. WADA prohibits GHRH analogs and growth hormone secretagogues under section S2.2.4. Seven of the eight compounds in this category fall under that section. IGF-1 LR3 falls under S2, and its insulin cross-activity also relates to S4. Check the live WADA list before any decision that involves competition.

01

Identity & Classification

GHRP-2 is also called pralmorelin. It is a synthetic hexapeptide growth hormone secretagogue.

02

Mechanism: How It Works

GHRP-2 acts as an agonist at the ghrelin receptor (GHS-R1a) on somatotrophs. It releases growth hormone and suppresses somatostatin.

It raises cortisol and prolactin by a moderate amount. It also stimulates appetite.

03

Research Findings

Evidence tier: clinical.

GHRP-2 increases food intake in healthy men (Laferrère et al., 2005).

It is a validated diagnostic agent for growth hormone deficiency.

04

Reported Dosing & Delivery

Japan approves a single-dose diagnostic use.

Any other dosing is non-clinical. No clinical source supports it.

05

Pharmacokinetics & Timing

The half-life is short.

06

Interactions & Combinations

GHRP-2 acts on the growth hormone axis. Community combinations are anecdotal.

Compare with GHRP-6 and Ipamorelin.

07

Safety & Contraindications

GHRP-2 causes a transient increase in cortisol and prolactin. It increases appetite.

Single diagnostic doses were generally well tolerated. Repeated-dose safety data are limited.

08

Monitoring & Biomarkers

  • IGF-1.
  • Glucose.
  • Cortisol.
09

Regulatory Status

Japan approves pralmorelin as a single-dose diagnostic. The FDA does not approve it.

WADA prohibits it under S2.2.4. See the WADA prohibited list status.

10

Works Cited

  1. 01Laferrère B, et al. Journal of Clinical Endocrinology & Metabolism, 2005. PMID 15699539. View source →
  2. 02Drugs in R&D, 2004. PMID 15230633. View source →
  3. 03WADA 2025 Prohibited List, section S2.2.4. View source →

Research use only. This is not medical advice.

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